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- Table of Contents
Information about Atypical Teratoid/rhabdoid Tumor: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Most recent studies have shown that Atypical Teratoid/rhabdoid Tumor shares some biological mechanisms with brain-neoplasms, carcinoma, central-nervous-system-neoplasms, cerebellar-neoplasms, ependymoma, glioma, malignant-neoplasms, malignant-paraganglionic-neoplasm, medulloblastoma, meningioma, monosomy, neoplasm-metastasis, neoplasms, nervous-system-neoplasms, nervousness, neuroectodermal-tumor-primitive, neuroectodermal-tumors, rhabdoid-tumor, teratoma.
Among the many pathways, these few ones have gauged particular interests from scientists studying Atypical Teratoid/rhabdoid Tumor, and have been seen in publications frequently: Angiogenesis, Atp-dependent Chromatin Remodeling, Cell Cycle, Cell Cycle Arrest, Cell Growth, Cell Killing, Cell Proliferation, Cellular Process, Chromatin Remodeling, Fertilization, Induction Of Apoptosis, Interphase, Localization, Methylation, Mitosis, Pathogenesis, Response To Irinotecan, Rna Interference, S Phase, Senescence
Quite a number of genes have been found to play important roles in Atypical Teratoid/rhabdoid Tumor, such as CD99, CDKN2A, CLDN6, DES, ETFA, GFAP, IGF2, LAMC2, MB, MID1, MUC1, MYC, POMT1, POU5F1, SMARCB1, SPP1, SYP, TP53, VIM. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.