Disease Info Card

Neuroectodermal Tumors

Information about Neuroectodermal Tumors: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.

Overview of Neuroectodermal Tumors

Most recent studies have shown that Neuroectodermal Tumors shares some biological mechanisms with astrocytoma, bone-neoplasms, brain-neoplasms, carcinoma, cerebellar-neoplasms, ependymoma, ewings-sarcoma-primitive-neuroectodermal-tumor-(pnet), glioma, malignant-neoplasms, malignant-paraganglionic-neoplasm, medulloblastoma, neoplasm-metastasis, neoplasms, neoplasms-germ-cell-and-embryonal, nervousness, neuroblastoma, neuroectodermal-tumor-melanotic, neuroectodermal-tumor-primitive, rhabdomyosarcoma, sarcoma.

Among the many pathways, these few ones have gauged particular interests from scientists studying Neuroectodermal Tumors, and have been seen in publications frequently: Angiogenesis, Cell Adhesion, Cell Cycle, Cell Death, Cell Differentiation, Cell Growth, Cell Migration, Cell Proliferation, Enucleation, Immune Response, Interphase, Localization, Metaphase, Methylation, Oncogenesis, Pathogenesis, Reverse Transcription, Secretion, Transport

Quite a number of genes have been found to play important roles in Neuroectodermal Tumors, such as CD99, CTLA4, DES, ENO1, ENO2, EWSR1, FLI1, FLII, GFAP, HLA-DQA1, MB, MYC, MYCN, NOD2, SMARCB1, SS18L1, SYP, TP53, VIM. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.

Neuroectodermal Tumors Related Genes

click to see detail information for each gene

CD99 CTLA4 DES
ENO1 ENO2 EWSR1
FLI1 FLII GFAP
HLA-DQA1 MB MYC
MYCN NOD2 SMARCB1
SS18L1 SYP TP53
VIM