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Disease Info Card
Amyloid Neuropathies, Familial
Information about Amyloid Neuropathies, Familial: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Amyloid Neuropathies, Familial
Most recent studies have shown that Amyloid Neuropathies, Familial shares some biological mechanisms with adenomatous-polyposis-coli, alzheimers-disease, amyloid-deposition, amyloid-neuropathies, amyloid-polyneuropathy-type-i, amyloidosis, amyloidosis-familial, cardiomyopathies, carpal-tunnel-syndrome, nervous-system-disorder, nervousness, peripheral-neuropathy, plaque-amyloid, polyneuropathy, primary-systemic-amyloidosis, senile-cardiac-amyloidosis.
Among the many pathways, these few ones have gauged particular interests from scientists studying Amyloid Neuropathies, Familial, and have been seen in publications frequently: Aging, Amyloid Fibril Formation, Cardiac Conduction, Coagulation, Excretion, Gastric Emptying, Gene Conversion, Glomerular Filtration, Innervation, Localization, Pathogenesis, Protein Folding, Proteolysis, Reflex, Regeneration, Retinol Transport, Rna Interference, Secretion, Transport, Tropism
Quite a number of genes have been found to play important roles in Amyloid Neuropathies, Familial, such as ALB, APCS, CALR, CDAN1, CEL, CSF2, EPO, FAP, GLMN, GSN, LAMC2, LTA, MPZ, PMP22, RBP4, RENBP, TEAD1, TRIM21, TTR. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.