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Disease Info Card
Senile Cardiac Amyloidosis
Information about Senile Cardiac Amyloidosis: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Senile Cardiac Amyloidosis
Most recent studies have shown that Senile Cardiac Amyloidosis shares some biological mechanisms with amyloid-deposition, amyloid-neuropathies-familial, amyloidosis, amyloidosis-familial, cardiac-arrhythmia, cardiomyopathies, heart-diseases, heart-failure, hypertrophic-cardiomyopathy, hypertrophy, left-ventricular-hypertrophy, multiple-myeloma, plaque-amyloid, polyneuropathy, primary-amyloidosis, primary-systemic-amyloidosis, restrictive-cardiomyopathy.
Among the many pathways, these few ones have gauged particular interests from scientists studying Senile Cardiac Amyloidosis, and have been seen in publications frequently: Aging, Amyloid Fibril Formation, Angiogenesis, Cardiac Conduction, Cell Differentiation, Circadian Rhythm, Coagulation, Diuresis, Excretion, Glomerular Filtration, Innervation, Localization, Lymphocyte Activation, Pathogenesis, Proteolysis, Senescence, Transport, Vasodilation
Quite a number of genes have been found to play important roles in Senile Cardiac Amyloidosis, such as ALB, APCS, APOA1, CALR, CEL, EMB, FAP, GLMN, IGKV1-5, LYZ, NPPB, TEAD1, TRIM21, TTR, XPO1. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.