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Disease Info Card
Allan-herndon-dudley Syndrome (ahds)
Information about Allan-herndon-dudley Syndrome (ahds): characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Allan-herndon-dudley Syndrome (ahds)
Most recent studies have shown that Allan-herndon-dudley Syndrome (ahds) shares some biological mechanisms with ataxia, athetosis, developmental-disabilities, dysarthria, dyskinesia-paroxysmal, dyskinetic-syndrome, hypoplasia, hypothyroidism, muscle-contracture, muscle-hypotonia, muscle-spasticity, muscular-atrophy, myopathy, nervousness, paraplegia, severe-mental-retardation, spastic-paraplegia.
Among the many pathways, these few ones have gauged particular interests from scientists studying Allan-herndon-dudley Syndrome (ahds), and have been seen in publications frequently: Anion Transport, Brain Development, Chondrocyte Differentiation, Hormone Secretion, Hormone Transport, Myelination, Organic Anion Transport, Protein Folding, Regeneration, Secretion, Sex Determination, Thyroid Hormone Transport, Translation, Transmembrane Transport, Transport
Quite a number of genes have been found to play important roles in Allan-herndon-dudley Syndrome (ahds), such as BBS9, DIO2, IBSP, LAT2, LMOD1, PGK1, SLC16A10, SLC16A2, SLC25A5, SLC7A5, SLC7A7, SLC7A8, SLCO1C1, STS, TH, THRSP, TRH, TTR, VDAC1. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.