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Disease Info Card
Dyskinesia, Paroxysmal
Information about Dyskinesia, Paroxysmal: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Dyskinesia, Paroxysmal
Most recent studies have shown that Dyskinesia, Paroxysmal shares some biological mechanisms with absence-epilepsy, ataxia, athetosis, channelopathies, chorea, choreoathetosis, convulsions, dyskinetic-syndrome, dystonia-disorders, epilepsy, involuntary-movements, migraine-disorders, movement-disorders, multiple-sclerosis, nervous-system-disorder, paroxysmal-choreoathetosis, paroxysmal-dystonia, paroxysmal-nonkinesigenic-dyskinesia, sclerosis.
Among the many pathways, these few ones have gauged particular interests from scientists studying Dyskinesia, Paroxysmal, and have been seen in publications frequently: Aging, Anion Transport, Brain Development, Calcium Ion Homeostasis, Cell Aging, Cell Death, Cellular Localization, Exocytosis, Glucose Transport, Ion Homeostasis, Localization, Long-term Synaptic Potentiation, Myelination, Pathogenesis, Reflex, Regeneration, Response To Stress, Synaptic Transmission, Transport, Vesicle Docking
Quite a number of genes have been found to play important roles in Dyskinesia, Paroxysmal, such as CACNA1A, CAMP, CSF2, FGF14, KCNMA1, LAMC2, OCA2, PDC, PEA15, PNKD, PRKD1, PRRT2, SLC16A2, SLC2A1, SLC4A3, TOR1A. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.