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Disease Info Card
Acth Hypersecretion
Information about Acth Hypersecretion: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Acth Hypersecretion
Most recent studies have shown that Acth Hypersecretion shares some biological mechanisms with acth-dependent-cushings-syndrome, acth-secreting-pituitary-adenoma, acth-syndrome-ectopic, addison-disease, adenoma, adrenal-cortical-adenoma, adrenal-gland-neoplasms, adrenal-hyperplasia, carcinoid-tumor, carcinoma, congenital-adrenal-hyperplasia, cushing-syndrome, hyperplasia, hypoglycemia, nelson-syndrome, neoplasms, pituitary-adenoma, pituitary-dependent-cushings-disease, pituitary-diseases, pituitary-neoplasms.
Among the many pathways, these few ones have gauged particular interests from scientists studying Acth Hypersecretion, and have been seen in publications frequently: Blood Coagulation, Cell Proliferation, Circadian Rhythm, Coagulation, Cortisol Secretion, Excretion, Hormone Secretion, Hypersensitivity, Localization, Membrane Depolarization, Pathogenesis, Peptide Hormone Processing, Peptide Secretion, Reflex, Regeneration, Response To Stress, Secretion, Secretory Pathway, Tube Formation
Quite a number of genes have been found to play important roles in Acth Hypersecretion, such as AVP, C1QL1, CBX4, CRH, CS, GGH, GH1, GIP, HPSE, INS, KRT17, KRT6B, PBX1, PCSK2, PKD2, POMC, PPARG, PRL, SST. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.