pathway Info Card

Uterine Smooth Muscle Contraction

Information about Uterine Smooth Muscle Contraction: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.

Overview of Uterine Smooth Muscle Contraction

Most recent studies have shown that Uterine Smooth Muscle Contraction shares some biological mechanisms with cell-growth, cell-proliferation, cellular-process, embryo-implantation, lactation, luteinization, menstruation, milk-ejection, muscle-contraction, natriuresis, neuroprotection, parturition, progesterone-secretion, reflex, regulation-of-cell-growth, regulation-of-uterine-smooth-muscle-contraction, secretion, smooth-muscle-contraction, transport, vasoconstriction.

Among the many pathways, these few ones have gauged particular interests from scientists studying Uterine Smooth Muscle Contraction, and have been seen in publications frequently: cell-growth, cell-proliferation, cellular-process, embryo-implantation, lactation, luteinization, menstruation, milk-ejection, muscle-contraction, natriuresis, neuroprotection, parturition, progesterone-secretion, reflex, regulation-of-cell-growth, regulation-of-uterine-smooth-muscle-contraction, secretion, smooth-muscle-contraction, transport, vasoconstriction

Quite a number of genes have been found to play important roles in Uterine Smooth Muscle Contraction, such as BBS9, CAT, EDN1, EDN3, EDNRA, EDNRB, GSR, KNG1, Kcnk4, MS4A1, MYL2, MYLK, Myh14, NMU, OXT, OXTR, PGF, PLEKHM1, SPANXB1. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this pathway. Plesae stay updated.