Disease Info Card

Ureteral Obstruction

Information about Ureteral Obstruction: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.

Overview of Ureteral Obstruction

Most recent studies have shown that Ureteral Obstruction shares some biological mechanisms with fibrosis, gastroesophageal-reflux-disease, hydronephrosis, infection-of-kidney, kidney-calculi, kidney-diseases, kidney-failure, malignant-neoplasms, neoplasms, obstructive-uropathy, pain, pathological-dilatation, retroperitoneal-fibrosis, stenosis, ureteral-calculi, ureteral-diseases, ureteropelvic-junction-obstruction-nos, urinary-tract-infection, urologic-diseases, vesico-ureteral-reflux.

Among the many pathways, these few ones have gauged particular interests from scientists studying Ureteral Obstruction, and have been seen in publications frequently: Cell Death, Cell Proliferation, Coagulation, Diuresis, Excretion, Fibroblast Proliferation, Glomerular Filtration, Inflammatory Response, Innervation, Localization, Micturition, Natriuresis, Pathogenesis, Peristalsis, Regeneration, Secretion, Transport, Transposition, Tubuloglomerular Feedback, Vasoconstriction

Quite a number of genes have been found to play important roles in Ureteral Obstruction, such as ACE, AGT, CCL2, EGF, FN1, HGF, NOS2, PCNA, RAPGEF5, REN, SLC22A3, SMAD2, SMAD3, TGFB1, TNF. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.