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Disease Info Card
Tumor Escape
Information about Tumor Escape: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Tumor Escape
Most recent studies have shown that Tumor Escape shares some biological mechanisms with adenocarcinoma, carcinoma, cell-transformation-neoplastic, decreased-immunologic-activity-[pe], inflammation, leukemia, lung-neoplasms, lymphoma, malignant-neoplasms, malignant-paraganglionic-neoplasm, malignant-squamous-cell-neoplasm, melanoma, neoplasm-metastasis, neoplasms, neoplasms-experimental, pathologic-neovascularization, skin-neoplasms, tumor-angiogenesis, tumor-progression.
Among the many pathways, these few ones have gauged particular interests from scientists studying Tumor Escape, and have been seen in publications frequently: Angiogenesis, Cell Activation, Cell Adhesion, Cell Cycle, Cell Death, Cell Differentiation, Cell Growth, Cell Killing, Cell Proliferation, Cell Recognition, Cytokine Production, Cytolysis, Immune Response, Induction Of Apoptosis, Oncogenesis, Pathogenesis, Secretion, T Cell Activation, T Cell Proliferation, Translation
Quite a number of genes have been found to play important roles in Tumor Escape, such as CD274, CD4, CD80, CD8A, CTLA4, DCX, ERBB2, FAS, FASLG, FOXP3, HLA-DQA1, HLA-E, IFNG, IL10, IL2, KLRK1, NOD2, PDCD1, TNF, VEGFA. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.