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Disease Info Card
Transient Cerebral Ischemia
Information about Transient Cerebral Ischemia: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Transient Cerebral Ischemia
Most recent studies have shown that Transient Cerebral Ischemia shares some biological mechanisms with arterial-occlusion, brain-injuries, brain-ischemia, carotid-artery-occlusion, cerebral-artery-occlusion, cerebral-infarction, cerebral-ischemia, cerebrovascular-accident, cerebrovascular-disorders, edema, infarction, infarction-middle-cerebral-artery, ischemia, ischemic-stroke, nerve-degeneration, occlusion-middle-cerebral-artery, reperfusion-injury, transient-ischemia, transient-ischemic-attack.
Among the many pathways, these few ones have gauged particular interests from scientists studying Transient Cerebral Ischemia, and have been seen in publications frequently: Aging, Cell Adhesion, Cell Death, Cell Proliferation, Cellular Localization, Coagulation, Hyperphosphorylation, Immune Response, Inflammatory Response, Localization, Neurogenesis, Neuroprotection, Pathogenesis, Platelet Aggregation, Programmed Cell Death, Response To Ischemia, Synaptic Transmission, Translation, Transport, Vasoconstriction
Quite a number of genes have been found to play important roles in Transient Cerebral Ischemia, such as BCL2, BCL2L1, BDNF, CA1, CA3, CASP3, CAT, GAD1, GFAP, GLUL, MAP2, NOS1, NOS2, NOS3, ODC1, SOD1, TNF. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.