Disease Info Card

Thrombocythemia, Essential

Information about Thrombocythemia, Essential: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.

Overview of Thrombocythemia, Essential

Most recent studies have shown that Thrombocythemia, Essential shares some biological mechanisms with acute-leukemia, acute-megakaryocytic-leukemias, anemia, dysmyelopoietic-syndromes, fibrosis, hemorrhage, leukemia, leukemia-myelocytic-acute, malignant-neoplasms, metaplasia, myeloid-leukemia, myeloid-leukemia-chronic, myeloproliferative-disease, neoplasms, polycythemia, polycythemia-vera, primary-myelofibrosis, reactive-thrombocytosis, thrombocytosis, thrombosis.

Among the many pathways, these few ones have gauged particular interests from scientists studying Thrombocythemia, Essential, and have been seen in publications frequently: Angiogenesis, Blood Coagulation, Cell Cycle, Cell Growth, Cell Proliferation, Coagulation, Dna Methylation, Excretion, Fibrinolysis, Hemopoiesis, Hemostasis, Hypersensitivity, Interphase, Methylation, Pathogenesis, Platelet Activation, Platelet Aggregation, Reverse Transcription, Secretion, Translation

Quite a number of genes have been found to play important roles in Thrombocythemia, Essential, such as ABL1, ASXL1, BCR, CD177, CD34, EPO, F2, IL3, JAK2, MPL, MTTP, PAFAH1B1, PF4, SF3B1, THPO, TIMP1, TPO, VWF, YWHAE. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.

Thrombocythemia, Essential Related Genes

click to see detail information for each gene

ABL1 ASXL1 BCR
CD177 CD34 EPO
F2 IL3 JAK2
MPL MTTP PAFAH1B1
PF4 SF3B1 THPO
TIMP1 TPO VWF
YWHAE