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Disease Info Card
Thermal Hyperalgesia
Information about Thermal Hyperalgesia: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Thermal Hyperalgesia
Most recent studies have shown that Thermal Hyperalgesia shares some biological mechanisms with chronic-pain, diabetes-mellitus, diabetes-mellitus-experimental, diabetic-neuropathies, edema, hyperalgesia, hyperalgesia-tactile, hyperalgesias-mechanical, inflammation, nerve-damage, nervousness, neuralgia, pain, peripheral-nerve-injuries, peripheral-neuropathy, sciatic-neuropathy, stenosis.
Among the many pathways, these few ones have gauged particular interests from scientists studying Thermal Hyperalgesia, and have been seen in publications frequently: Astrocyte Activation, Cell Activation, Cell Death, Hypersensitivity, Immune Response, Inflammatory Response, Innervation, Localization, Mast Cell Degranulation, Pathogenesis, Reflex, Regeneration, Response To Heat, Secretion, Sensitization, Swimming, Synaptic Transmission, Transport, Vasoconstriction, Vasodilation
Quite a number of genes have been found to play important roles in Thermal Hyperalgesia, such as BDNF, CALCA, FOS, GFAP, IL6, KNG1, NGF, NOS1, NOS2, PRRT2, PTGS2, RPL5, S100A12, TAC1, TBCA, TNF, TRPV1. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.