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- Table of Contents
Information about Tangier Disease: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Most recent studies have shown that Tangier Disease shares some biological mechanisms with abetalipoproteinemia, arteriosclerosis, atherosclerosis, blood-protein-disorders, cardiovascular-diseases, coronary-artery-disease, coronary-heart-disease, diabetes-mellitus, heart-diseases, hereditary-diseases, high-density-lipoprotein-deficiency, hypercholesterolemia, hyperlipidemia, hyperlipoproteinemia-type-iib, hyperlipoproteinemias, hypoalphalipoproteinemia-familial, hypolipoproteinemias, lecithin-acyltransferase-deficiency, peripheral-neuropathy.
Among the many pathways, these few ones have gauged particular interests from scientists studying Tangier Disease, and have been seen in publications frequently: Aging, Cell Proliferation, Cholesterol Efflux, Cholesterol Esterification, Cholesterol Homeostasis, Cholesterol Transport, Excretion, Lipid Binding, Lipid Homeostasis, Lipid Storage, Lipid Transport, Localization, Pathogenesis, Phospholipid Efflux, Proteolysis, Reverse Cholesterol Transport, Secretion, Secretory Pathway, Translation, Transport
Quite a number of genes have been found to play important roles in Tangier Disease, such as ABCA1, ABCA4, ABCB6, APOA1, APOA2, APOA4, APOC3, ATP6V0A1, CETP, LCAT, LPA, LPL, PLTP, RFC1, RFC2, RFC4, SCARB1. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.