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Disease Info Card
Subacute Thyroiditis
Information about Subacute Thyroiditis: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Subacute Thyroiditis
Most recent studies have shown that Subacute Thyroiditis shares some biological mechanisms with adenoma, autoimmune-diseases, autoimmune-reaction, carcinoma, goiter, graves-disease, hashimoto-disease, hyperthyroidism, hypothyroidism, inflammation, malignant-neoplasm-of-thyroid, malignant-neoplasms, nodular-goiter, pain, thyroid-diseases, thyroid-neoplasm, thyroid-nodule, thyroiditis, thyrotoxicosis.
Among the many pathways, these few ones have gauged particular interests from scientists studying Subacute Thyroiditis, and have been seen in publications frequently: Angiogenesis, Calcitonin Secretion, Cell Activation, Cell Adhesion, Cell Growth, Excretion, Fibroblast Proliferation, Hypersensitivity, Immune Response, Leukocyte Migration, Localization, Pathogenesis, Proteolysis, Reflex, Regeneration, Secretion, Sensitization, T Cell Activation, Tissue Regeneration, Transport
Quite a number of genes have been found to play important roles in Subacute Thyroiditis, such as ALB, CALCA, CRP, CSRP1, ESR1, HLA-DRB4, IL2, IL6, POMC, SAT1, SERPINA7, SHBG, SLC25A5, TG, THPO, TNF, TPO, TRH, TSHR, VEGFA. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.