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- Table of Contents
Information about Subacute Cutaneous Lupus Erythematosus: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Most recent studies have shown that Subacute Cutaneous Lupus Erythematosus shares some biological mechanisms with arthritis, autoimmune-diseases, autoimmune-reaction, dermatitis, dermatologic-disorders, drug-eruptions, erythema, exanthema, heart-block, lupus-erythematosus-cutaneous, lupus-erythematosus-discoid, lupus-erythematosus-systemic, malignant-neoplasms, neonatal-lupus-erythematosus, photosensitivity-disorders, rheumatism, rheumatoid-arthritis, sjogrens-syndrome, vasculitis.
Among the many pathways, these few ones have gauged particular interests from scientists studying Subacute Cutaneous Lupus Erythematosus, and have been seen in publications frequently: Antibody-dependent Cellular Cytotoxicity, Cell Activation, Cell Adhesion, Cell Differentiation, Chemotaxis, Coagulation, Demethylation, Dna Demethylation, Dna Methylation, Exocytosis, Hypersensitivity, Immune Response, Localization, Methylation, Neutrophil Chemotaxis, Pathogenesis, Photoprotection, Protein Secretion, Secretion, T Cell Activation
Quite a number of genes have been found to play important roles in Subacute Cutaneous Lupus Erythematosus, such as C2, C3, C4A, CALR, CD4, CD8A, CTLA4, FUT3, HNRNPC, HPS4, ICAM1, NOD2, SSB, TNF, TNFRSF25, TRIM21. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.