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Information about Spinal Muscular Atrophies Of Childhood: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Most recent studies have shown that Spinal Muscular Atrophies Of Childhood shares some biological mechanisms with atrophy, dystrophy, flexed-fetal-attitude, hmn-(hereditary-motor-neuropathy)-proximal-type-i, juvenile-spinal-muscular-atrophy, monomelic-amyotrophy, motor-neuron-disease, muscle-hypotonia, muscle-weakness, muscular-atrophy, muscular-atrophy-spinal-type-ii, muscular-dystrophy, myopathy, neuromuscular-diseases, respiratory-failure, scoliosis-unspecified, spinal-muscular-atrophy, weakness.
Among the many pathways, these few ones have gauged particular interests from scientists studying Spinal Muscular Atrophies Of Childhood, and have been seen in publications frequently: Cell Adhesion, Cell Death, Cell Proliferation, Cognition, Dna Repair, Gene Conversion, Glycosylation, Innervation, Localization, Muscle Atrophy, Muscle Contraction, Neuron Death, Pathogenesis, Programmed Cell Death, Regeneration, Rna Processing, Rna Splicing, Skeletal Muscle Atrophy, Translation, Transport
Quite a number of genes have been found to play important roles in Spinal Muscular Atrophies Of Childhood, such as BCL2, C7, CXCL10, CYCS, DMD, GRIP1, GTF2H2, HLA-E, IGFALS, IGHMBP2, MAP1B, NAIP, PSMC6, SMN1, SNRPN, SOD1, STMN1. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.