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Disease Info Card
Pupil Disorders
Information about Pupil Disorders: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Pupil Disorders
Most recent studies have shown that Pupil Disorders shares some biological mechanisms with angle-closure-glaucoma, autonomic-nervous-system-disorders, blind-vision, cataract, corneal-diseases, diabetes-mellitus, disorder-of-the-optic-nerve, eye-abnormalities, glaucoma, hemorrhage, intraocular-pressure-disorder, iris-diseases, mydriasis, nervousness, ocular-ophthalmoplegia-sympathetic, retinal-diseases, stenosis.
Among the many pathways, these few ones have gauged particular interests from scientists studying Pupil Disorders, and have been seen in publications frequently: Circadian Rhythm, Coagulation, Enucleation, Excretion, Eye Development, Fibrinolysis, Flight, Glomerular Filtration, Hypersensitivity, Innervation, Localization, Micturition, Pathogenesis, Reflex, Regeneration, Response To Cocaine, Response To Light Stimulus, Secretion, Synaptic Transmission, Transport
Quite a number of genes have been found to play important roles in Pupil Disorders, such as ACD, ACE, ALB, BRCA1, ERG, GCLC, KCNH2, LAMB2, LAMC1, LMNB2, MID1, SDS, SLC25A5, TNFSF14, UGCG. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.