Disease Info Card

Proliferative Inflammatory Atrophy

Information about Proliferative Inflammatory Atrophy: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.

Overview of Proliferative Inflammatory Atrophy

Most recent studies have shown that Proliferative Inflammatory Atrophy shares some biological mechanisms with arthritis, atrophy, brain-neoplasms, edema, hemorrhage, infarction, infective-disorder, inflammation, ischemia, malignant-neoplasm-of-prostate, malignant-neoplasms, meningeal-disorder, neoplasms, nervousness, pain, pertussis, prostatic-neoplasms, rheumatoid-arthritis, staphylococcal-infections, tissue-adhesions.

Among the many pathways, these few ones have gauged particular interests from scientists studying Proliferative Inflammatory Atrophy, and have been seen in publications frequently: Aging, Angiogenesis, Biofilm Formation, Cell Cycle, Cell Death, Cell Growth, Cell Migration, Cell Proliferation, Glucose Transport, Immune Response, Inflammatory Response, Innervation, Localization, Pathogenesis, Phagocytosis, Regeneration, Secretion, Synaptic Transmission, Transport, Virulence

Quite a number of genes have been found to play important roles in Proliferative Inflammatory Atrophy, such as ADA, ADORA1, AQP4, ATP6V0A1, ATP6V0A2, CAT, CRAT, CSF2, GFAP, GLYAT, GPHA2, INS, LAMC2, NLRP5, RFC1, RFC2, RFC4, RICTOR, SGCA, TNF. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.