This website uses cookies to ensure you get the best experience on our website.
- Table of Contents
, No Gclids
Disease Info Card
Persistent Hyperplastic Primary Vitreous
Information about Persistent Hyperplastic Primary Vitreous: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Persistent Hyperplastic Primary Vitreous
Most recent studies have shown that Persistent Hyperplastic Primary Vitreous shares some biological mechanisms with cataract, congenital-abnormality, congenital-ocular-coloboma-(disorder), corneal-diseases, disorder-of-eye, dysplasia, eye-abnormalities, eye-neoplasms, glaucoma, hemorrhage, hyperplasia, microphthalmos, neoplasms, retinal-detachment, retinal-diseases, retinal-dysplasia, retinoblastoma, retinopathy-of-prematurity.
Among the many pathways, these few ones have gauged particular interests from scientists studying Persistent Hyperplastic Primary Vitreous, and have been seen in publications frequently: Angiogenesis, Cell Cycle, Cell Cycle Arrest, Cell Death, Cell Proliferation, Coagulation, Embryo Development, Enucleation, Eye Development, Fermentation, Hemostasis, Inflammatory Response, Localization, Myelination, Pathogenesis, Pigmentation, Reflex, Secretion, Tissue Morphogenesis, Vasculature Development
Quite a number of genes have been found to play important roles in Persistent Hyperplastic Primary Vitreous, such as ATOH7, CDKN2A, CDKN2D, DDX4, ELSPBP1, FH, FOXC1, GFAP, IKBKG, LBR, OPN1LW, PAX6, PITX2, REG1A, TGFB1, TP53, VEGFA. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.