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Disease Info Card
Peptic Ulcer Hemorrhage
Information about Peptic Ulcer Hemorrhage: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Peptic Ulcer Hemorrhage
Most recent studies have shown that Peptic Ulcer Hemorrhage shares some biological mechanisms with acute-hemorrhage, duodenal-ulcer, duodenal-ulcer-hemorrhage, gastric-ulcer, gastritis, gastroduodenal-haemorrhage, gastrointestinal-hemorrhage, helicobacter-infections, hematemesis, hemorrhage, massive-hemorrhage, melena, peptic-ulcer, peptic-ulcer-perforation, physiological-stress, stomach-neoplasms, ulcer, ulcer-haemorrhage, upper-gastrointestinal-hemorrhage.
Among the many pathways, these few ones have gauged particular interests from scientists studying Peptic Ulcer Hemorrhage, and have been seen in publications frequently: Acid Secretion, Aging, Blood Coagulation, Coagulation, Excretion, Fibrinolysis, Gastric Acid Secretion, Gastric Emptying, Hemostasis, Inflammatory Response, Localization, Mucus Secretion, Pathogenesis, Platelet Aggregation, Proteolysis, Regeneration, Secretion, Transport, Vasoconstriction, Virulence
Quite a number of genes have been found to play important roles in Peptic Ulcer Hemorrhage, such as ABO, ALB, CLIP1, CLIP2, DLC1, GAST, GNAI1, GNL3, HP, KRAS, PGA3, PGA4, PGA5, POMC, PTGS2, SCT, SST. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.