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Disease Info Card
Upper Gastrointestinal Hemorrhage
Information about Upper Gastrointestinal Hemorrhage: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Upper Gastrointestinal Hemorrhage
Most recent studies have shown that Upper Gastrointestinal Hemorrhage shares some biological mechanisms with acute-upper-gastrointestinal-hemorrhage, duodenal-ulcer, esophageal-varix, gastric-ulcer, gastritis, gastrointestinal-hemorrhage, hematemesis, hemorrhage, hypertension-portal, hypertensive-disease, liver-cirrhosis, malignant-neoplasms, melena, neoplasms, pain, peptic-ulcer, peptic-ulcer-hemorrhage, ulcer, varicosity.
Among the many pathways, these few ones have gauged particular interests from scientists studying Upper Gastrointestinal Hemorrhage, and have been seen in publications frequently: Acid Secretion, Aging, Blood Coagulation, Circadian Rhythm, Coagulation, Dehiscence, Excretion, Fibrinolysis, Gastric Acid Secretion, Gastric Emptying, Hemostasis, Inflammatory Response, Localization, Pathogenesis, Pigmentation, Platelet Aggregation, Secretion, Transposition, Vasoconstriction, Wound Healing
Quite a number of genes have been found to play important roles in Upper Gastrointestinal Hemorrhage, such as ALB, ARSA, COX5A, COX8A, CPOX, DLC1, EVL, F2, GAST, GNAI1, GNL3, HP, KIT, POMC, PTGS2, SLC17A5, SST. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.