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Disease Info Card
Overactive Bladder
Information about Overactive Bladder: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Overactive Bladder
Most recent studies have shown that Overactive Bladder shares some biological mechanisms with benign-prostatic-hypertrophy, bladder-neck-obstruction, constipation, hyperplasia, lower-urinary-tract-symptoms, nervousness, neurogenic-urinary-bladder, nocturia, pain, prostatic-hyperplasia, urge-incontinence, urinary-bladder-diseases, urinary-incontinence, urinary-stress-incontinence, urinary-tract-infection, urination-disorders, xerostomia.
Among the many pathways, these few ones have gauged particular interests from scientists studying Overactive Bladder, and have been seen in publications frequently: Aging, Cell Proliferation, Cognition, Defecation, Diuresis, Excretion, Hypersensitivity, Innervation, Localization, Menopause, Micturition, Muscle Contraction, Pathogenesis, Reflex, Regeneration, Secretion, Sensitization, Smooth Muscle Contraction, Translation, Transport
Quite a number of genes have been found to play important roles in Overactive Bladder, such as AKR1B1, CASK, CHRM1, CYP3A4, EFS, GJA1, KLK3, MCC, NGF, P2RX3, PSMD3, PVR, RANGAP1, RPS3, SLC17A5, TAC1, TRPV1. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.