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Disease Info Card
Osteopoikilosis (disorder)
Information about Osteopoikilosis (disorder): characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Osteopoikilosis (disorder)
Most recent studies have shown that Osteopoikilosis (disorder) shares some biological mechanisms with albers-schonberg-disease, bone-diseases, bone-diseases-developmental, bone-neoplasms, connective-tissue-diseases, connective-tissue-nevus-nos, cytogenetic-abnormality, dermatofibrosis-lenticularis-disseminata, dermatologic-disorders, dysplasia, fibroma, melanocytic-nevus, melorheostosis, metastatic-malignant-neoplasm-to-the-bone, neoplasm-metastasis, osteosclerosis, pain, sclerosis, skin-neoplasms.
Among the many pathways, these few ones have gauged particular interests from scientists studying Osteopoikilosis (disorder), and have been seen in publications frequently: Bone Remodeling, Bone Resorption, Cell Proliferation, Innervation, Intramembranous Ossification, Localization, Ossification, Osteoblast Differentiation, Osteoclast Development, Osteoclast Differentiation, Pathogenesis, Segmentation, Tissue Morphogenesis, Wound Healing
Quite a number of genes have been found to play important roles in Osteopoikilosis (disorder), such as ELN, ENO1, EXT1, GRIP1, HMGA2, ITGB3, LEMD3, LGALS12, MB, MID1, NCOA2, NF1, POMC, SMAD2, SS18L1. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.