Disease Info Card

Old Myocardial Infarction

Information about Old Myocardial Infarction: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.

Overview of Old Myocardial Infarction

Most recent studies have shown that Old Myocardial Infarction shares some biological mechanisms with acute-myocardial-infarction, angina-pectoris, cardiac-arrhythmia, cardiac-fibrillation, cardiomyopathies, cardiomyopathy-dilated, coronary-artery-disease, coronary-heart-disease, heart-diseases, heart-failure, hypertensive-disease, hypertrophy, infarction, ischemia, myocardial-infarction, myocardial-ischemia, stenosis, sudden-death, tachycardia-ventricular, ventricular-dysfunction-left.

Among the many pathways, these few ones have gauged particular interests from scientists studying Old Myocardial Infarction, and have been seen in publications frequently: Aging, Angiogenesis, Cardiac Conduction, Cell Adhesion, Cell Death, Cell Differentiation, Coagulation, Endothelial Cell Proliferation, Fibrinolysis, Hibernation, Inflammatory Response, Innervation, Localization, Pathogenesis, Platelet Aggregation, Reflex, Reverse Transcription, Secretion, Transposition, Vasodilation

Quite a number of genes have been found to play important roles in Old Myocardial Infarction, such as ACE, DLD, FUT2, GNL3, HTRA2, ITGB2, KRAS, LAD1, LPA, MITF, NDUFB6, NPPB, PREP, REST. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.