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- Table of Contents
Information about Nodular Lymphocyte Predominant Hodgkin Lymphoma: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Most recent studies have shown that Nodular Lymphocyte Predominant Hodgkin Lymphoma shares some biological mechanisms with b-cell-lymphomas, burkitt-lymphoma, cell-transformation-neoplastic, classical-hodgkins-lymphoma, diffuse-large-b-cell-lymphoma, hodgkin-disease, hyperplasia, ki-1--anaplastic-large-cell-lymphoma, lymphocyte-rich-classical-hodgkin-lymphoma, lymphoma, lymphoma-follicular, lymphoma-non-hodgkin, lymphoproliferative-disorders, malignant-neoplasms, malignant-paraganglionic-neoplasm, neoplasms, nodular-sclerosis-classical-hodgkin-lymphoma, sclerosis, t-cell-histiocyte-rich-large-b-cell-lymphoma, t-cell-lymphoma.
Among the many pathways, these few ones have gauged particular interests from scientists studying Nodular Lymphocyte Predominant Hodgkin Lymphoma, and have been seen in publications frequently: Cell Cycle, Cell Cycle Phase, Cell Differentiation, Cell Proliferation, Coagulation, Demethylation, Immune Response, Induction Of Apoptosis, Inflammatory Response, Interphase, Localization, Metaphase, Mitosis, Nuclear Export, Oncogenesis, Pathogenesis, Somatic Hypermutation Of Immunoglobulin Genes, T Cell Differentiation
Quite a number of genes have been found to play important roles in Nodular Lymphocyte Predominant Hodgkin Lymphoma, such as B3GAT1, BCL2, BCL6, CD79A, CTLA4, FUT4, HES1, HLA-DQA1, HMGCL, KRT20, LIPC, MID1, MS4A1, NOD2, PAX5, PDCD1, POU2F2, PTPRC, TNFRSF8. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.