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Disease Info Card
Juvenile Neuronal Ceroid Lipfuscinosis
Information about Juvenile Neuronal Ceroid Lipfuscinosis: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Juvenile Neuronal Ceroid Lipfuscinosis
Most recent studies have shown that Juvenile Neuronal Ceroid Lipfuscinosis shares some biological mechanisms with atrophy, blind-vision, cerebral-atrophy, dementia, dog-diseases, epilepsy, infantile-neuronal-ceroid-lipofuscinosis, late-infantile-neuronal-ceroid-lipfuscinosis, lipoidosis, lysosomal-storage-diseases, nerve-degeneration, nervous-system-disorder, nervousness, neurodegenerative-disorders, neuronal-ceroid-lipofuscinoses, retinal-degeneration, storage-disease, visual-impairment.
Among the many pathways, these few ones have gauged particular interests from scientists studying Juvenile Neuronal Ceroid Lipfuscinosis, and have been seen in publications frequently: Aging, Arginine Transport, Autophagy, Brain Development, Cell Activation, Cell Death, Cell Growth, Cytokinesis, Endocytosis, Glycosylation, Immune Response, Localization, Macroautophagy, Myelination, Neuron Death, Pathogenesis, Proteolysis, Response To Oxidative Stress, Translation, Transport
Quite a number of genes have been found to play important roles in Juvenile Neuronal Ceroid Lipfuscinosis, such as CLN3, CLN5, CLN6, CLN8, CTSD, CTSH, DNAJC5, ERG, EXOSC10, GAD1, GAD2, GLUL, HP, KCNH2, NCL, PPT1, SULT1A1, TPP1. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.