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Disease Info Card
Multiple Transition Abnormalities
Information about Multiple Transition Abnormalities: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Multiple Transition Abnormalities
Most recent studies have shown that Multiple Transition Abnormalities shares some biological mechanisms with adenocarcinoma, adenoma, brain-neoplasms, carcinogenesis, carcinoma, cell-transformation-neoplastic, colonic-neoplasms, colorectal-cancer, colorectal-neoplasms, lung-neoplasms, lymphoma, malignant-neoplasms, malignant-paraganglionic-neoplasm, malignant-squamous-cell-neoplasm, multiple-transversion-abnormalities, mutation-out-of-frame, neoplasms, skin-neoplasms, transition-mutation.
Among the many pathways, these few ones have gauged particular interests from scientists studying Multiple Transition Abnormalities, and have been seen in publications frequently: Cell Cycle, Cell Death, Demethylation, Dna Alkylation, Dna Methylation, Dna Repair, Dna Replication, Drug Resistance, Gene Conversion, Gene Silencing, Immune Response, Methylation, Mismatch Repair, Pathogenesis, Proteolysis, Reverse Transcription, S Phase, Sos Response, Translesion Synthesis, Viral Replication
Quite a number of genes have been found to play important roles in Multiple Transition Abnormalities, such as ADA, AGFG1, AGT, AGXT, AICDA, APC, APOBEC3B, CDA, GLB1, HPRT1, HRAS, KRAS, MGMT, RIPK1, RPAIN, TP53, UNG. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.