Disease Info Card

Exocrine Pancreatic Insufficiency

Information about Exocrine Pancreatic Insufficiency: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.

Overview of Exocrine Pancreatic Insufficiency

Most recent studies have shown that Exocrine Pancreatic Insufficiency shares some biological mechanisms with atrophy, bone-marrow-diseases, celiac-disease, cystic-fibrosis, diabetes-mellitus, diabetes-mellitus-insulin-dependent, diarrhea, dog-diseases, fibrosis, malabsorption-syndrome, malignant-neoplasms, malnutrition, pain, pancreatic-diseases, pancreatic-insufficiency, pancreatic-neoplasm, pancreatitis, pancreatitis-chronic, shwachman-syndrome, steatorrhea.

Among the many pathways, these few ones have gauged particular interests from scientists studying Exocrine Pancreatic Insufficiency, and have been seen in publications frequently: Acid Secretion, Cell Death, Chemotaxis, Chromosome Breakage, Defecation, Excretion, Gastric Acid Secretion, Gastric Emptying, Gene Conversion, Glucagon Secretion, Insulin Secretion, Intestinal Absorption, Lipid Digestion, Localization, Neutrophil Chemotaxis, Pathogenesis, Ribosome Biogenesis, Secretion, Translation, Transport

Quite a number of genes have been found to play important roles in Exocrine Pancreatic Insufficiency, such as ALB, CCK, CELA1, CELA3A, CELA3B, CFTR, CP, GCG, GIP, GLB1, INS, PNLIP, PPY, RIPK2, SBDS, SCT, UBR1. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.