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- Table of Contents
Information about Medullary Sponge Kidney: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Most recent studies have shown that Medullary Sponge Kidney shares some biological mechanisms with acidosis, congenital-hemihypertrophy, cystic-disease--retired-, cystic-kidney-diseases, fibrosis, hypercalciuria, hypertrophy, infection-of-kidney, kidney-calculi, kidney-diseases, kidney-neoplasm, liver-cirrhosis, nephrocalcinosis, nephrolithiasis, pathological-dilatation, polycystic-kidney-diseases, renal-tubular-acidosis, tuberculosis-renal, urinary-tract-infection.
Among the many pathways, these few ones have gauged particular interests from scientists studying Medullary Sponge Kidney, and have been seen in publications frequently: Bone Mineralization, Cell Proliferation, Excretion, Flight, Glomerular Filtration, Intestinal Absorption, Lactation, Localization, Micturition, Muscle Atrophy, Natriuresis, Parathyroid Hormone Secretion, Pathogenesis, Response To Acid, Secretion, Testosterone Secretion, Transport, Transposition, Vasodilation, Water Transport
Quite a number of genes have been found to play important roles in Medullary Sponge Kidney, such as AMBP, ARTN, GDNF, GLO1, HOPX, INS, KRT7, LYZ, NTF3, PKHD1, PLOD1, PSPN, PTH, RAPGEF5, REN, RET, SIK1, STXBP3, UMOD. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.