Disease Info Card

Leydig Cell Hyperplasia

Information about Leydig Cell Hyperplasia: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.

Overview of Leydig Cell Hyperplasia

Most recent studies have shown that Leydig Cell Hyperplasia shares some biological mechanisms with adenoma, atrophy, carcinoma, cell-transformation-neoplastic, cryptorchidism, fibrosis, gynecomastia, hyperplasia, hypertrophy, hypogonadism, infertility, leydig-cell-tumor, male-infertility, malignant-neoplasms, neoplasms, ovarian-neoplasm, pituitary-diseases, precocious-puberty, testicular-diseases, testicular-neoplasms.

Among the many pathways, these few ones have gauged particular interests from scientists studying Leydig Cell Hyperplasia, and have been seen in publications frequently: Aging, Cell Cycle, Cell Development, Cell Differentiation, Cell Growth, Cell Proliferation, Copulation, Enucleation, Excretion, Gonad Development, Gonadotropin Secretion, Leydig Cell Differentiation, Localization, Mating, Pathogenesis, Secretion, Sperm Motility, Spermatogenesis, Testosterone Secretion, Transport

Quite a number of genes have been found to play important roles in Leydig Cell Hyperplasia, such as AKR1B1, AMH, AR, AREG, BRD2, CYP19A1, DBP, FDXR, FST, GC, HSD17B4, LHCGR, MAPK3, NR0B1, PCNA, PLOD1, POMC, PRL. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.