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Disease Info Card
Lambert-eaton Myasthenic Syndrome
Information about Lambert-eaton Myasthenic Syndrome: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Lambert-eaton Myasthenic Syndrome
Most recent studies have shown that Lambert-eaton Myasthenic Syndrome shares some biological mechanisms with autoimmune-diseases, autoimmune-reaction, carcinoma, lung-neoplasms, malignant-neoplasm-of-lung, malignant-neoplasms, muscle-weakness, myasthenia-gravis, myasthenias, myopathy, neoplasms, nervous-system-disorder, nervousness, neuromuscular-diseases, paraneoplastic-syndromes, small-cell-carcinoma-of-lung, weakness.
Among the many pathways, these few ones have gauged particular interests from scientists studying Lambert-eaton Myasthenic Syndrome, and have been seen in publications frequently: Aging, Cell Adhesion, Complement Activation, Endocytosis, Exocytosis, Glycosylation, Hormone Secretion, Hypersensitivity, Immune Response, Innervation, Localization, Membrane Depolarization, Muscle Contraction, Neuromuscular Synaptic Transmission, Pathogenesis, Phagocytosis, Reflex, Secretion, Synaptic Transmission, Transport
Quite a number of genes have been found to play important roles in Lambert-eaton Myasthenic Syndrome, such as ACHE, AMPH, AVP, BCHE, CACNA1A, CDR2, CSF2, CST7, DAP, DBNL, DMPK, DNPEP, DPYSL5, LAMC2, OAT, POMC, SOX1, SYT1. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.