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- Table of Contents
Information about Juvenile Hyaline Fibromatosis: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Most recent studies have shown that Juvenile Hyaline Fibromatosis shares some biological mechanisms with beckwith-wiedemann-syndrome, dysplasia, fetal-growth-retardation, fibroma, fibromatosis, fibromatosis-gingival, flexed-fetal-attitude, gingival-hyperplasia, gingival-hypertrophy, hyalinization, hyalinosis-systemic, hydatidiform-mole, hyperplasia, hypertrophy, muscle-contracture, neoplasms, partial-hydatidiform-mole, placenta-disorders, skin-neoplasms.
Among the many pathways, these few ones have gauged particular interests from scientists studying Juvenile Hyaline Fibromatosis, and have been seen in publications frequently: Aging, Bone Maturation, Cell Division, Cell Proliferation, Coagulation, Cytokinesis, Fertilization, Gland Morphogenesis, Hemostasis, Localization, Lymphangiogenesis, Mammary Gland Development, Mammary Gland Morphogenesis, Methylation, Pathogenesis, Pigmentation, Platelet Aggregation, Spinal Cord Development, Tissue Development, Translation
Quite a number of genes have been found to play important roles in Juvenile Hyaline Fibromatosis, such as AMY2A, ANTXR1, ANTXR2, BLOC1S6, CDKN1C, CHM, CORO1A, EPB42, GLI1, MPZ, PRH1, PTCH1, TIMP1, VIM, VWF. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.