Disease Info Card

Gingival Hypertrophy

Information about Gingival Hypertrophy: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.

Overview of Gingival Hypertrophy

Most recent studies have shown that Gingival Hypertrophy shares some biological mechanisms with dental-plaque, dermatologic-disorders, edema, epilepsy, fibroma, fibromatosis, fibromatosis-gingival, gingival-diseases, gingival-hyperplasia, gingivitis, hemorrhage, hyperplasia, hypertrichosis, hypertrophy, inflammation, juvenile-hyaline-fibromatosis, leukemia, muscle-contracture, neoplasms, periodontal-diseases.

Among the many pathways, these few ones have gauged particular interests from scientists studying Gingival Hypertrophy, and have been seen in publications frequently: Aging, Bone Resorption, Coagulation, Fibrinolysis, Fibroblast Proliferation, Glomerular Filtration, Hemostasis, Hypersensitivity, Interleukin-2 Production, Keratinization, Lactation, Localization, Mastication, Pathogenesis, Pigmentation, Response To Hypoxia, Tooth Eruption, Translation, Vasoconstriction, Wound Healing

Quite a number of genes have been found to play important roles in Gingival Hypertrophy, such as ANTXR2, AVP, CD7, CSH1, ERCC8, FANCB, HSPA9, IL6, MMP2, MMP9, MPO, NCAM1, PPOX, RAPGEF5, SERPINA3, TNF, VIM. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.