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Disease Info Card
Hereditary Motor And Sensory Neuropathies
Information about Hereditary Motor And Sensory Neuropathies: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Hereditary Motor And Sensory Neuropathies
Most recent studies have shown that Hereditary Motor And Sensory Neuropathies shares some biological mechanisms with atrophy, charcot-marie-tooth-disease, dejerine-sottas-disease-(disorder), demyelinating-diseases, demyelination, hereditary-liability-to-pressure-palsies, hereditary-motor-and-sensory-neuropathy-type-i, hereditary-sensory-and-autonomic-neuropathies, hypertrophy, inherited-neuropathy, muscle-weakness, muscular-atrophy, nervousness, neuromuscular-diseases, peripheral-neuropathy, polyneuropathy, sensory-neuropathy, spinal-muscular-atrophy, weakness.
Among the many pathways, these few ones have gauged particular interests from scientists studying Hereditary Motor And Sensory Neuropathies, and have been seen in publications frequently: Cell Adhesion, Cell Death, Cell Proliferation, Glycosylation, Innervation, Localization, Meiosis, Mitochondrial Fission, Muscle Atrophy, Muscle Hypertrophy, Myelin Maintenance, Myelination, Ossification, Pathogenesis, Reflex, Regeneration, Rna Processing, Schwann Cell Proliferation, Translation, Transport
Quite a number of genes have been found to play important roles in Hereditary Motor And Sensory Neuropathies, such as BSCL2, EGR2, GDAP1, GDI1, GJB1, HSPB1, KIF1B, MAD2L1BP, MFN2, MPZ, MTMR2, NDRG1, PES1, PMP22, PXMP2, RPLP0, SBF2, SLC12A6, TRPV4. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.