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- Table of Contents
Information about Dejerine-sottas Disease (disorder): characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Most recent studies have shown that Dejerine-sottas Disease (disorder) shares some biological mechanisms with ataxia, atrophy, charcot-marie-tooth-disease, congenital-hypomyelinating-neuropathy, demyelinating-diseases, demyelination, hereditary-liability-to-pressure-palsies, hereditary-motor-and-sensory-neuropathies, hereditary-motor-and-sensory-neuropathy-type-i, hypertrophy, inherited-neuropathy, muscular-atrophy, nervousness, peripheral-neuropathy, polyneuropathy, sensory-neuropathy, weakness.
Among the many pathways, these few ones have gauged particular interests from scientists studying Dejerine-sottas Disease (disorder), and have been seen in publications frequently: Cell Adhesion, Cell Death, Cell Growth, Cell Proliferation, Fertilization, Glycosylation, Localization, Meiosis, Muscle Atrophy, Myelin Maintenance, Myelination, Pathogenesis, Proprioception, Reflex, Regeneration, Regulation Of Cell Proliferation, Regulation Of Myelination, Schwann Cell Proliferation, Single Strand Break Repair, Translation
Quite a number of genes have been found to play important roles in Dejerine-sottas Disease (disorder), such as EGR2, GDAP1, GDI1, GJB1, KIF1B, MAD2L1BP, MFN2, MPZ, MTMR2, NR0B1, PMP22, PRB4, PRDX6, PXMP2, RPLP0, SBF2. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.