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Disease Info Card
Fracture Of Talus
Information about Fracture Of Talus: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Fracture Of Talus
Most recent studies have shown that Fracture Of Talus shares some biological mechanisms with carcinoma, cardiovascular-diseases, diabetes-mellitus, diabetes-mellitus-experimental, diabetic-nephropathy, dislocations, edema, fibrosis, fracture, inflammation, ischemia, kidney-diseases, malignant-neoplasms, myocarditis, neoplasms, plant-poisoning, poisoning, polyendocrinopathies-autoimmune, reperfusion-injury, sheep-diseases.
Among the many pathways, these few ones have gauged particular interests from scientists studying Fracture Of Talus, and have been seen in publications frequently: Aging, Angiogenesis, Blood Circulation, Cell Cycle, Cell Differentiation, Cell Growth, Cell Migration, Cell Proliferation, Cytokine Production, Excretion, Germination, Immune Response, Lymphocyte Proliferation, Nodulation, Pathogenesis, Regeneration, S Phase, Secretion, Senescence, Wound Healing
Quite a number of genes have been found to play important roles in Fracture Of Talus, such as ACE, ALB, BCL2, CD4, CD8A, IFNG, IL10, IL2, IL4, IL6, INS, KLK3, MAPK1, NFKB1, NOS2, SH2B2, SLC17A5, SOD1, TNF, VEGFA. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.