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Disease Info Card
Fetal Macrosomia
Information about Fetal Macrosomia: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Fetal Macrosomia
Most recent studies have shown that Fetal Macrosomia shares some biological mechanisms with birth-injuries, congenital-abnormality, diabetes-mellitus, diabetes-mellitus-insulin-dependent, diabetes-mellitus-non-insulin-dependent, fetal-death, fetal-growth-retardation, gestational-diabetes, hyperglycemia, hyperinsulinism, hypertensive-disease, hypoglycemia, impaired-glucose-tolerance, obesity, obstetric-labor-complications, pre-eclampsia, pregnancy-complications, pregnancy-in-diabetics, shoulder-dystocia.
Among the many pathways, these few ones have gauged particular interests from scientists studying Fetal Macrosomia, and have been seen in publications frequently: Cell Growth, Cell Proliferation, Dehiscence, Excretion, Fertilization, Gluconeogenesis, Glucose Homeostasis, Glucose Transport, Insulin Secretion, Lactation, Lipid Transport, Localization, Menarche, Menopause, Ovulation, Parturition, Pathogenesis, Reflex, Secretion, Transport
Quite a number of genes have been found to play important roles in Fetal Macrosomia, such as ADA, ADIPOQ, AGA, ASAH1, CS, GLS2, GPC3, HNF1A, HNF4A, IGF1, IGF2, IGFBP3, INS, LEP, NDUFB6, RANGAP1, RFC1, RFC2, SLC17A5. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.