Shoulder Dystocia

Overview of Shoulder Dystocia

Most recent studies have shown that Shoulder Dystocia shares some biological mechanisms with birth-injuries, brachial-plexus-injury, brachial-plexus-neuropathies, congenital-abnormality, diabetes-mellitus, erb-duchenne-paralysis, fetal-macrosomia, fracture, gestational-diabetes, hemorrhage, hypertensive-disease, hypoglycemia, laceration, obesity, obstetric-labor-complications, postpartum-hemorrhage, pre-eclampsia, pregnancy-complications, pregnancy-in-diabetics.

Among the many pathways, these few ones have gauged particular interests from scientists studying Shoulder Dystocia, and have been seen in publications frequently: Dehiscence, Embryo Development, Inflammatory Response, Insulin Secretion, Locomotion, Parturition, Proprioception, Reflex, Regeneration, Secretion, System Development, Translation

Quite a number of genes have been found to play important roles in Shoulder Dystocia, such as ASAH1, CALM3, CDH11, CS, DMPK, ESR2, GNL3, GPHA2, HAS1, INS, LEP, NDUFB6, NTF3, PICALM, PRG4, RANGAP1, SGCA, SLC17A5, SNAP91. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.

Shoulder Dystocia Related Genes

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Pathways Related to Shoulder Dystocia

This information is being compiled and will come in a future update

Related Diseases