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Disease Info Card
Familial Hypercholesterolemia - Heterozygous
Information about Familial Hypercholesterolemia - Heterozygous: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Familial Hypercholesterolemia - Heterozygous
Most recent studies have shown that Familial Hypercholesterolemia - Heterozygous shares some biological mechanisms with arteriosclerosis, atherosclerosis, cardiovascular-diseases, coronary-arteriosclerosis, coronary-artery-disease, coronary-heart-disease, diabetes-mellitus, dyslipidemias, heart-diseases, hypercholesterolemia, hypercholesterolemia-familial, hyperlipidemia, hyperlipoproteinemia-type-iib, hyperlipoproteinemias, hypertensive-disease, infarction, myocardial-infarction, stenosis, xanthoma, xanthomatosis.
Among the many pathways, these few ones have gauged particular interests from scientists studying Familial Hypercholesterolemia - Heterozygous, and have been seen in publications frequently: Aging, Cholesterol Esterification, Cholesterol Homeostasis, Cholesterol Transport, Coagulation, Complement Activation, Dna Amplification, Excretion, Gallstone Formation, Inflammatory Response, Lipid Oxidation, Localization, Pathogenesis, Platelet Aggregation, Response To Lipopolysaccharide, Response To Statin, Reverse Cholesterol Transport, Secretion, Transport, Vasodilation
Quite a number of genes have been found to play important roles in Familial Hypercholesterolemia - Heterozygous, such as APOA1, APOB, APOE, CD55, CETP, CFH, CRP, FH, HMGCR, INS, LDLR, LPA, PCSK9, SEPSECS, TCN2, TG, TNF. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.