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Disease Info Card
Esophageal Atresia
Information about Esophageal Atresia: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Esophageal Atresia
Most recent studies have shown that Esophageal Atresia shares some biological mechanisms with anastomotic-leak, anus-imperforate, atresia, congenital-abnormality, congenital-heart-defects, deglutition-disorders, diaphragmatic-hernia, esophageal-atresia-with-tracheo-esophageal-fistula, esophageal-diseases, esophageal-fistula, esophageal-stenosis, gastroesophageal-reflux-disease, hernia, intestinal-atresia, pathologic-fistula, pathological-dilatation, pneumonia, stenosis, tracheoesophageal-fistula.
Among the many pathways, these few ones have gauged particular interests from scientists studying Esophageal Atresia, and have been seen in publications frequently: Cell Adhesion, Cell Proliferation, Dehiscence, Foregut Morphogenesis, Gastric Emptying, Gastric Motility, Innervation, Localization, Lung Development, Lung Morphogenesis, Notochord Development, Pathogenesis, Peristalsis, Reflex, Rumination, Secretion, Segmentation, Transport, Transposition, Wound Healing
Quite a number of genes have been found to play important roles in Esophageal Atresia, such as AFP, C2, CALCA, CHD7, CRP, ELSPBP1, FEZF2, FGF1, FGF10, FGF7, GNAI1, MYCN, NKX2-1, NOG, SHH, SOX2, SS18L1, TEF, UCHL1. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.