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Disease Info Card
Ectrodactyly
Information about Ectrodactyly: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Ectrodactyly
Most recent studies have shown that Ectrodactyly shares some biological mechanisms with aplasia-nos, cleft-lip, cleft-palate, cleft-palate-with-cleft-lip, congenital-abnormality, congenital-absence, congenital-foot-deformity, congenital-hand-deformities, cytogenetic-abnormality, dysplasia, ectodermal-dysplasia, ectrodactyly-ectodermal-dysplasia-clefting-syndrome, hypoplasia, limb-defect, limb-deformities-congenital, polydactyly, syndactyly, teratogenic-effect.
Among the many pathways, these few ones have gauged particular interests from scientists studying Ectrodactyly, and have been seen in publications frequently: Cell Death, Cell Proliferation, Cellular Homeostasis, Cellular Senescence, Dna Alkylation, Electron Transport, Hair Follicle Morphogenesis, Limb Development, Limb Morphogenesis, Localization, Locomotion, Micturition, Nipple Development, Ossification, Osteoblast Differentiation, Pathogenesis, Pigmentation, Translation, Transport, Transposition
Quite a number of genes have been found to play important roles in Ectrodactyly, such as CDH3, CKAP4, DLX5, DLX6, FBXW4, FGF4, FGF8, GLI2, NOD2, PAEP, RPE65, SHH, SNX3, TP53, TP63, UVRAG. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.