Dominant Hyperferritinemia And Cataract

Overview of Dominant Hyperferritinemia And Cataract

Most recent studies have shown that Dominant Hyperferritinemia And Cataract shares some biological mechanisms with anemia, autoimmune-diseases, bilateral-cataracts-(disorder), cataract, de-novo-mutation, eye-diseases-hereditary, hemochromatosis, hereditary-diseases, hereditary-hemochromatosis, inflammation, iron-deficiency, iron-deficiency-anemia, iron-metabolism-disorders, iron-overload, lens-opacities, malignant-neoplasms, nuclear-cataract, senile-cataract.

Among the many pathways, these few ones have gauged particular interests from scientists studying Dominant Hyperferritinemia And Cataract, and have been seen in publications frequently: Dna Amplification, Glycosylation, Hypersensitivity, Localization, Pathogenesis, Phagocytosis, Secretion, Translation, Transport

Quite a number of genes have been found to play important roles in Dominant Hyperferritinemia And Cataract, such as ACO1, AGRP, CP, FTH1, FTL, GZMA, HAMP, HFE, IREB2, TBC1D2, TF, TFR2, WNT2. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.

Dominant Hyperferritinemia And Cataract Related Genes

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Pathways Related to Dominant Hyperferritinemia And Cataract

This information is being compiled and will come in a future update

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