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Disease Info Card
Bilateral Cataracts (disorder)
Information about Bilateral Cataracts (disorder): characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Bilateral Cataracts (disorder)
Most recent studies have shown that Bilateral Cataracts (disorder) shares some biological mechanisms with ataxia, atrophy, blind-vision, cataract, diabetes-mellitus, dystrophy, edema, glaucoma, intraocular-lymphoma, muscle-hypotonia, nystagmus, retinal-detachment, strabismus, visual-impairment, werner-syndrome, xanthomatosis, xanthomatosis-cerebrotendinous.
Among the many pathways, these few ones have gauged particular interests from scientists studying Bilateral Cataracts (disorder), and have been seen in publications frequently: Aging, Brain Development, Cell Adhesion, Cell Differentiation, Coagulation, Electron Transport, Electron Transport Chain, Excretion, Glycosylation, Lipid Storage, Muscle Atrophy, Ossification, Oxidative Phosphorylation, Pathogenesis, Pigmentation, Programmed Cell Death, Proteolysis, Reflex, Translation, Transport
Quite a number of genes have been found to play important roles in Bilateral Cataracts (disorder), such as CDKN2A, CYP27A1, DMPK, DNM2, ERG, FTL, GALK1, GALT, GJA8, INS, KCNH2, PTH, RANGAP1, SORD, TF. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.