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Disease Info Card
Senile Cataract
Information about Senile Cataract: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Senile Cataract
Most recent studies have shown that Senile Cataract shares some biological mechanisms with age-related-macular-degeneration, aqueous-humor-disorders, blind-vision, capsule-opacification, cataract, cataract-subcapsular, corneal-diseases, diabetes-mellitus, disorder-of-eye, glaucoma, glaucoma-open-angle, intraocular-lymphoma, intraocular-pressure-disorder, lens-opacities, nuclear-cataract, posterior-capsule-opacification, retinal-diseases, visual-impairment.
Among the many pathways, these few ones have gauged particular interests from scientists studying Senile Cataract, and have been seen in publications frequently: Aging, Cell Death, Cell Proliferation, Cellular Homeostasis, Dehiscence, Dna Repair, Epithelial Cell Proliferation, Glycosylation, Inflammatory Response, Localization, Menopause, Methylation, Muscle Contraction, Pathogenesis, Pigmentation, Protein Oxidation, Reflex, Senescence, Transport, Wound Healing
Quite a number of genes have been found to play important roles in Senile Cataract, such as ACD, AKR1B1, ALB, CAT, CRYAA, G6PD, GSR, GSTM1, IL6, LAMA3, MIP, NDUFB6, NOL3, PHEX, SOD1, TNFSF14. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.