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Disease Info Card
Iron Overload
Information about Iron Overload: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Iron Overload
Most recent studies have shown that Iron Overload shares some biological mechanisms with anemia, anemia-sickle-cell, beta-thalassemia, cooleys-anemia, diabetes-mellitus, fibrosis, hemochromatosis, hemosiderosis, hepatitis, hepatitis-c, hereditary-hemochromatosis, infective-disorder, inflammation, iron-deficiency, liver-carcinoma, liver-cirrhosis, liver-diseases, malignant-neoplasms, siderosis, thalassemia.
Among the many pathways, these few ones have gauged particular interests from scientists studying Iron Overload, and have been seen in publications frequently: Aging, Cell Cycle, Cell Death, Cell Proliferation, Coagulation, Endocytosis, Excretion, Immune Response, Insulin Secretion, Intestinal Absorption, Localization, Pathogenesis, Phagocytosis, Pigmentation, Protein Oxidation, Regeneration, Secretion, Translation, Transport, Virulence
Quite a number of genes have been found to play important roles in Iron Overload, such as CACNA1C, CP, EPO, FUT1, HAMP, HFE, IL6, INS, SLC11A2, SLC17A5, SLC40A1, TBXAS1, TF, TFR2, TYMS. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.