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Disease Info Card
Lens Opacities
Information about Lens Opacities: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.
Overview of Lens Opacities
Most recent studies have shown that Lens Opacities shares some biological mechanisms with blind-vision, cataract, cataract-subcapsular, corneal-diseases, diabetes-mellitus, diabetic-retinopathy, disorder-of-eye, glaucoma, hemorrhage, intraocular-pressure-disorder, myopia, nuclear-cataract, refractive-errors, retinal-detachment, retinal-diseases, sclerosis, senile-cataract, visual-impairment.
Among the many pathways, these few ones have gauged particular interests from scientists studying Lens Opacities, and have been seen in publications frequently: Aging, Cell Differentiation, Cell Migration, Coagulation, Dna Repair, Enucleation, Flight, Lactation, Localization, Menarche, Menopause, Pathogenesis, Pigmentation, Protein Phosphorylation, Proteolysis, Reflex, Regeneration, Secretion, Translation, Transport
Quite a number of genes have been found to play important roles in Lens Opacities, such as AKR1B1, ALB, CAT, COX5A, CRYAA, DMPK, GALK1, GJA8, GLA, HMGCR, INS, LAMA3, NDUFB6, NF2, TNFSF14. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.
In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.