Disease Info Card

Congenital Elevation Of Scapula

Information about Congenital Elevation Of Scapula: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.

Overview of Congenital Elevation Of Scapula

Most recent studies have shown that Congenital Elevation Of Scapula shares some biological mechanisms with acquired-kyphosis, basal-cell-nevus-syndrome, cervical-rib, congenital-abnormal-synostosis, congenital-abnormality, congenital-musculoskeletal-anomalies, dislocations, dysplasia, fetal-growth-retardation, flexed-fetal-attitude, hemivertebra, hypoplasia, klippel-feil-syndrome, movement-disorders, orbital-separation-excessive, pain, scoliosis-unspecified, spina-bifida, spina-bifida-occulta.

Among the many pathways, these few ones have gauged particular interests from scientists studying Congenital Elevation Of Scapula, and have been seen in publications frequently: Menarche, Muscle Attachment, Ossification, Pathogenesis, Regeneration, Segmentation, Translation

Quite a number of genes have been found to play important roles in Congenital Elevation Of Scapula, such as ARC, ARHGAP4, BRD2, C2, C3, C5, C6, CCDC6, GDF6, HNRNPC, NOL3, PLOD1, PSMA7, PTCH1, RET, SS18L1, TAS2R38, TMCO1. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.

Congenital Elevation Of Scapula Related Genes

click to see detail information for each gene

ARC ARHGAP4 BRD2
C2 C3 C5
C6 CCDC6 GDF6
HNRNPC NOL3 PLOD1
PSMA7 PTCH1 RET
SS18L1 TAS2R38 TMCO1

Pathways Related to Congenital Elevation Of Scapula

This information is being compiled and will come in a future update

Menarche Muscle Attachment Ossification
Pathogenesis Regeneration Segmentation
Translation