Disease Info Card

Compression Of Optic Nerve

Information about Compression Of Optic Nerve: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.

Overview of Compression Of Optic Nerve

Most recent studies have shown that Compression Of Optic Nerve shares some biological mechanisms with blind-vision, diplopia, disorder-of-eye, disorder-of-the-optic-nerve, edema, exophthalmos, graves-disease, hematoma, hemorrhage, inflammation, intraocular-pressure-disorder, meningioma, neoplasms, nerve-compression, nerve-compression-syndrome, orbit-(eye-disorders), orbital-neoplasms, thyroid-associated-opthalmopathies, visual-impairment.

Among the many pathways, these few ones have gauged particular interests from scientists studying Compression Of Optic Nerve, and have been seen in publications frequently: Bone Resorption, Cell Death, Enucleation, Hemostasis, Innervation, Localization, Neuroprotection, Pathogenesis, Pigmentation, Reflex, Regeneration, Secretion

Quite a number of genes have been found to play important roles in Compression Of Optic Nerve, such as CAT, CRAT, CSF2, ERG, GFAP, GLYAT, GORAB, IL10, IL2, KCNH2, LAMC2, RFC1, RFC2, SH2D1A, TCIRG1. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.

Compression Of Optic Nerve Related Genes

click to see detail information for each gene

CAT CRAT CSF2
ERG GFAP GLYAT
GORAB IL10 IL2
KCNH2 LAMC2 RFC1
RFC2 SH2D1A TCIRG1

Pathways Related to Compression Of Optic Nerve

This information is being compiled and will come in a future update

Bone Resorption Cell Death Enucleation
Hemostasis Innervation Localization
Neuroprotection Pathogenesis Pigmentation
Reflex Regeneration Secretion