Disease Info Card

Cluster Headache

Information about Cluster Headache: characteristics, related genes and pathways, plus antibodies you can use for research. This page is being enriched constantly, if you see some information you would like this page to include please send your suggestions to us.

Overview of Cluster Headache

Most recent studies have shown that Cluster Headache shares some biological mechanisms with chronic-paroxysmal-hemicrania, common-migraine, conjunctival-hyperemia, depressive-disorder, episodic-cluster-headache, excessive-tearing, facial-pain, headache, headache-disorders, headache-disorders-primary, incomplete-anencephaly-hemicrania, migraine-disorders, nervousness, neuralgia, pain, paroxysmal-hemicrania, tension-headache, trigeminal-autonomic-cephalalgias, trigeminal-neuralgia, vascular-headaches.

Among the many pathways, these few ones have gauged particular interests from scientists studying Cluster Headache, and have been seen in publications frequently: Cell Activation, Circadian Rhythm, Cortisol Secretion, Excretion, Habituation, Hypersensitivity, Innervation, Localization, Mast Cell Degranulation, Menopause, Menstruation, Muscle Contraction, Pathogenesis, Platelet Aggregation, Reflex, Response To Pain, Secretion, Sensitization, Vasoconstriction, Vasodilation

Quite a number of genes have been found to play important roles in Cluster Headache, such as AURKA, C4B, CALCA, CD200R1, CRAT, CSF2, HNRNPC, IL2, LAMC2, LRP2, MCF2L, NLRP5, POMC, PRL, S100A12, SST, TAC1, TFF2, VIP. See what Boster has to offer for the research of these genes by clicking the gene name links below and view a more detailed info card/product listing for that gene.

In a later update, we will include information such as current drugs and therapy solutions as well as on-going and past clinical trials for this disease. Plesae stay updated.